Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. On the flip side, our capacity to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK signifies elevated FAK signaling downstream on the Pro32Pro33 mutant, as confirmed by elevated pFAK localization https://jackj788spk5.ja-blog.com/profile