We demonstrated that, in distinction to classical opioid receptors, ACKR3 will not result in classical G protein signaling and is not modulated via the classical prescription or analgesic opioids, like morphine, fentanyl, or buprenorphine, or by nonselective opioid antagonists which include naloxone. As an alternative, we established that LIH383, https://antonioa137tpc5.blogitright.com/profile